Similar articles for PMID: 21307261

Year	Number of Results
1993	3
1994	3
1996	1
1998	2
1999	1
2000	5
2001	10
2002	8
2003	7
2004	3
2005	4
2006	10
2007	7
2008	7
2009	10
2010	19
2011	19
2012	12
2013	19
2014	14
2015	8
2016	1
2017	5
2018	5
2019	4
2020	11
2021	11
2022	11
2023	3
2024	0

1

Membrane penetration by synaptotagmin is required for coupling calcium binding to vesicle fusion in vivo.

Paddock BE, Wang Z, Biela LM, Chen K, Getzy MD, Striegel A, Richmond JE, Chapman ER, Featherstone DE, Reist NE. Paddock BE, et al. J Neurosci. 2011 Feb 9;31(6):2248-57. doi: 10.1523/JNEUROSCI.3153-09.2011. J Neurosci. 2011. PMID: 21307261 Free PMC article.

2

Ca2+-dependent, phospholipid-binding residues of synaptotagmin are critical for excitation-secretion coupling in vivo.

Paddock BE, Striegel AR, Hui E, Chapman ER, Reist NE. Paddock BE, et al. J Neurosci. 2008 Jul 23;28(30):7458-66. doi: 10.1523/JNEUROSCI.0197-08.2008. J Neurosci. 2008. PMID: 18650324 Free PMC article.

3

synaptotagmin mutants reveal essential functions for the C2B domain in Ca2+-triggered fusion and recycling of synaptic vesicles in vivo.

Littleton JT, Bai J, Vyas B, Desai R, Baltus AE, Garment MB, Carlson SD, Ganetzky B, Chapman ER. Littleton JT, et al. J Neurosci. 2001 Mar 1;21(5):1421-33. doi: 10.1523/JNEUROSCI.21-05-01421.2001. J Neurosci. 2001. PMID: 11222632 Free PMC article.

4

The C2A domain of synaptotagmin is an essential component of the calcium sensor for synaptic transmission.

Bowers MR, Reist NE. Bowers MR, et al. PLoS One. 2020 Feb 7;15(2):e0228348. doi: 10.1371/journal.pone.0228348. eCollection 2020. PLoS One. 2020. PMID: 32032373 Free PMC article.

5

Drosophila synaptotagmin I null mutants show severe alterations in vesicle populations but calcium-binding motif mutants do not.

Loewen CA, Royer SM, Reist NE. Loewen CA, et al. J Comp Neurol. 2006 May 1;496(1):1-12. doi: 10.1002/cne.20868. J Comp Neurol. 2006. PMID: 16528727

6

Synaptotagmin I functions as a calcium sensor to synchronize neurotransmitter release.

Yoshihara M, Littleton JT. Yoshihara M, et al. Neuron. 2002 Dec 5;36(5):897-908. doi: 10.1016/s0896-6273(02)01065-6. Neuron. 2002. PMID: 12467593 Free article.

7

The C(2)B Ca(2+)-binding motif of synaptotagmin is required for synaptic transmission in vivo.

Mackler JM, Drummond JA, Loewen CA, Robinson IM, Reist NE. Mackler JM, et al. Nature. 2002 Jul 18;418(6895):340-4. doi: 10.1038/nature00846. Epub 2002 Jul 7. Nature. 2002. PMID: 12110842

8

Mutations in the second C2 domain of synaptotagmin disrupt synaptic transmission at Drosophila neuromuscular junctions.

Mackler JM, Reist NE. Mackler JM, et al. J Comp Neurol. 2001 Jul 16;436(1):4-16. J Comp Neurol. 2001. PMID: 11413542

9

Roles of SNARE proteins and synaptotagmin I in synaptic transmission: studies at the Drosophila neuromuscular synapse.

Kidokoro Y. Kidokoro Y. Neurosignals. 2003 Jan-Feb;12(1):13-30. doi: 10.1159/000068912. Neurosignals. 2003. PMID: 12624525 Free article. Review.

10

A synaptotagmin suppressor screen indicates SNARE binding controls the timing and Ca²⁺ cooperativity of vesicle fusion.

Guan Z, Bykhovskaia M, Jorquera RA, Sutton RB, Akbergenova Y, Littleton JT. Guan Z, et al. Elife. 2017 Sep 12;6:e28409. doi: 10.7554/eLife.28409. Elife. 2017. PMID: 28895532 Free PMC article.

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