Abstract
Can the superoxide radical exert deleterious effects independent of participating with H2O2 in the production of the hydroxyl radical? Examination of the superoxide-related literature reveals data suggesting an affirmative answer to this question.
MeSH terms
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Animals
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Catalase / metabolism
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Catalysis
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Chemotactic Factors / biosynthesis
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Erythrocytes / metabolism
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Ferritins / metabolism
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Glutathione Peroxidase / metabolism
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Hemoglobins / metabolism
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Inflammation / metabolism
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Isoenzymes / metabolism
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L-Lactate Dehydrogenase / metabolism
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Lipid Peroxides / physiology
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Lung / drug effects
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Macrophages / drug effects
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Molecular Weight
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Neutrophils / physiology
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Oxidation-Reduction
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Oxygen / toxicity
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Paraquat / toxicity
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Perfusion
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Peroxidase
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Peroxidases / metabolism
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Plants
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Saccharomyces cerevisiae / metabolism
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Shock, Hemorrhagic / metabolism
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Stem Cells / drug effects
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Superoxide Dismutase / metabolism
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Superoxides / physiology*
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Superoxides / toxicity
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Transketolase / metabolism
Substances
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Chemotactic Factors
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Hemoglobins
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Isoenzymes
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Lipid Peroxides
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Superoxides
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Ferritins
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L-Lactate Dehydrogenase
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Peroxidases
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pseudocatalase
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Catalase
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Peroxidase
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Glutathione Peroxidase
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Superoxide Dismutase
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Transketolase
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Paraquat
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Oxygen