Full PapersNeurites Can Remain Viable after Destruction of the Neuronal Soma by Programmed Cell Death (Apoptosis)
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Mechanisms and repair strategies for white matter degeneration in CNS injury and diseases
2021, Biochimica et Biophysica Acta - Molecular Basis of DiseaseAxon injury signaling and compartmentalized injury response in glaucoma
2019, Progress in Retinal and Eye ResearchCitation Excerpt :In the early 2000s, a theory was postulated suggesting axonal degeneration, rather than neuronal death, might be a primary driver of disease progression for some neurodegenerative conditions (Raff et al., 2002; Whitmore et al., 2005). Furthermore, work suggested that axons have unique molecular programs of degeneration which are independent from the molecular programs driving neuronal (somal) apoptosis (Deckwerth and Johnson, 1994; Finn et al., 2000; Whitmore et al., 2003). Using a wide variety of animal models of glaucoma, researchers have begun to identify and order the unique and overlapping molecules important for the somal and axonal programs of neurodegeneration in glaucomatous injury.
Neurite development and neurotoxicity
2018, Handbook of Developmental NeurotoxicologyBAX to basics: How the BCL2 gene family controls the death of retinal ganglion cells
2017, Progress in Retinal and Eye ResearchAxon degeneration: Context defines distinct pathways
2016, Current Opinion in Neurobiology
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